Dernière mise à jour le : 20 avril 2016

Prenatal exposure to persistent organic pollutants and organophosphate pesticides, and markers of glucose metabolism at birth

DESCRIPTION

Background :
Experimental evidence suggests that developmental exposure to persistent organic pollutants (POP) and to some non persistent pesticides may disrupt metabolic regulation of glucose metabolism and insulin secretion, and thereby contribute to the current epidemic of obesity and metabolic disorders. Quasi-experimental situations of undernutrition in utero have provided some information. However, the evidence in humans concerning the role of the prenatal environment in these disorders is contradictory, and little is known about long-term outcomes, such as type 2 diabetes, of prenatal exposure.

Objectives :
Our aim was to evaluate the effects of prenatal exposure to POP and organophosphate pesticides on fetal markers of glucose metabolism in a sample of newborns from the Pelagie mother–child cohort in Brittany (France).

NOTICE BIBLIOGRAPHIQUE DETAILLÉE

keyboard_arrow_right Titre : Prenatal exposure to persistent organic pollutants and organophosphate pesticides, and markers of glucose metabolism at birth
keyboard_arrow_right Type de documentation : Articles
keyboard_arrow_right Auteur(s) personne(s) : Cordier Sylvaine, Bonnet Fabrice, Rouget Florence, Monfort Christine, Bonvallot Nathalie, Chevrier Cécile, Massart Catherine, Warembourg Charline, Debost-Legrand Anne
keyboard_arrow_right Date de publication : 20 avril 2016
keyboard_arrow_right Editeur(s) : Elsevier
keyboard_arrow_right Tags thématiques : effet sur la santé, relation santé-environnement, risque pour la santé, pesticide, nourisson, pollution organique
keyboard_arrow_right Mots-clés libres : Adiponectin/Cord blood/Glucose metabolism markers/Insulin/Organophosphates/Persistent organic pollutants
keyboard_arrow_right Langue : Anglais
keyboard_arrow_right N° d'édition (ISBN,DOI...) : 10.1016/j.envres.2016.01.005
keyboard_arrow_right Collection : Environmental Research

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